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However, patients with isolated apical LVNC had lower LV end‐systolic diameter (39 versus 43 mm, P=0.012), greater right ventricular systolic pressure (28 versus 31 mm Hg, P=0.02), lower noncompacta thickness (17 versus 19 mm, P<0.0001), and lower NC:C ratio (2.6 versus 2.9, P=0.011).
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A significant proportion of DCM cases have an underlying genetic or inflammatory basis. Measurement of LV size and ejection fraction remain central to diagnosis, risk stratification, and treatment, but other aspects of cardiac remodeling inform prognosis and carry therapeutic implications.
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